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This is the first study measuring the serum TRX-1 levels in neonatal infants

Specifically, oxidative stress exacerbates endothelial dysfunction by damaging the vascular endothelium, causing ROS overproduction and AGE formation that impair vasodilation and promote chronic inflammation [2]

In parallel experiments, PDHA1 knockdown markedly enhanced the sensitivity of PCa cells to Elesclomol (ES), a potent inducer of cuproptosis, exerting a more pronounced cytotoxic effect than Enz

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ROCCs, receptor-operated calcium channels